All sections of society suffers from malnutrition. The poor lack the resources and the rich are succumbed to non-healthy options.
This results into acute immune disorders.
As a society, we are least bothered about our food sources. Food grown using chemicals is always lack of nutrition. And full of poisons.
Overall : प्राण विहीन inputs to body + प्राण consuming habits : Compromised cellular intelligence = reduced discrimination of self from others = immune disorders
Malnourished children are most likely to die from common infections, not starvation.
What’s also emerging is that the relationship between malnutrition and immune dysfunction may be a bit “chicken and egg,” with both causing and being the consequence of the other. Immune dysfunction results when people consume too few calories because of lack of food or have an excess of fat and sugar in their diet. That dysfunction is recorded in the DNA through epigenetic marks, so that if malnourished people have offspring, their children inherit an altered immune system (even after multiple generations). This altered immune system may then cause malnutrition even if children have an adequate diet.
In short, प्राण when compromised, body behaves unexpectedly.
Who cares? Work hard, party harder! 
😉 
😀 Future generation can go to hell! Why bother? 
(y) 
Research
Immune Dysfunction as a Cause and Consequence of Malnutrition
Trends
Undernourished children principally die of common infections, and immune defects are consistently demonstrated in under- and overnutrition.
Parental malnutrition leads to epigenetic modifications of infant immune and metabolic genes.
Healthy gut development relies on sensing of dietary nutrients, commensal, and pathogenic microbes via immune receptors.
Recurrent infections, chronic inflammation, and enteropathy compound clinical malnutrition by altering gut structure and function.
Immune cell activation and systemic proinflammatory mediator levels are increased in malnutrition.
Malnutrition impairs immune priming by DC and monocytes, and impairs effector memory T cell function.
Immune dysfunction can directly drive pathological processes in malnutrition, including malabsorption, increased metabolic demand, dysregulation of the growth hormone and HPA axes, and greater susceptibility to infection.
Malnutrition, which encompasses under- and overnutrition, is responsible for an enormous morbidity and mortality burden globally. Malnutrition results from disordered nutrient assimilation but is also characterized by recurrent infections and chronic inflammation, implying an underlying immune defect. Defects emerge before birth via modifications in the immunoepigenome of malnourished parents, and these may contribute to intergenerational cycles of malnutrition. This review summarizes key recent studies from experimental animals, in vitro models, and human cohorts, and proposes that immune dysfunction is both a cause and a consequence of malnutrition. Focusing on childhood undernutrition, we highlight gaps in current understanding of immune dysfunction in malnutrition, with a view to therapeutically targeting immune pathways as a novel means to reduce morbidity and mortality.
https://www.cell.com/trends/immunology/fulltext/S1471-4906(16)30006-0
