I recently delivered one speech where I discussed anti-aging based on what scriptures demands.
What is prescribed?
वन गमन – Go and live-in forest.
वेद पठन – Reciting Veda and performing meditation and tapas
What does it mean? To relax in resort like old age home. To listen old songs/bhajans on radio/iPod in your cozy apartment in resort? 
No! Forest life is most difficult life! It demands extra work from your body and brain. And that keeps you alive! And when you perform dhyan, meditation, tapas – you protect your hypothalamic glands in brain. The third eye of the head. And with healthy hypothalamus , aging is slowed down!
Last Week, I shared why primary education demands forest exposure for growing minds. Here is the old note I wrote about why we need forest exposure in old age!
The hypothalamus acts as the connector between the endocrine and nervous systems to achieve this. It plays a part in many essential functions of the body such as:
body temperature
thirst
appetite and weight control
emotions
sleep cycles
sex drive
childbirth
blood pressure and heart rate
production of digestive juices
balancing bodily fluids
Hypothalamus stem cells control aging.
वन गमन-वैराग्य-साधना-तप-जप = To me, all these activities after 50, are essential to maintain the Hypothalamus health. (by cortisol reduction i.e. stress hormone and by regular physical work in forest\farm)
Here is the paper talking about role of hypothalamic neural stem cells in aging.
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It has been proposed that the hypothalamus helps to control ageing, but the mechanisms responsible remain unclear. Here we develop several mouse models in which hypothalamic stem/progenitor cells that co-express Sox2 and Bmi1 are ablated, as we observed that ageing in mice started with a substantial loss of these hypothalamic cells. Each mouse model consistently displayed acceleration of ageing-like physiological changes or a shortened lifespan. Conversely, ageing retardation and lifespan extension were achieved in mid-aged mice that were locally implanted with healthy hypothalamic stem/progenitor cells that had been genetically engineered to survive in the ageing-related hypothalamic inflammatory microenvironment. Mechanistically, hypothalamic stem/progenitor cells contributed greatly to exosomal microRNAs (miRNAs) in the cerebrospinal fluid, and these exosomal miRNAs declined during ageing, whereas central treatment with healthy hypothalamic stem/progenitor cell-secreted exosomes led to the slowing of ageing. In conclusion, ageing speed is substantially controlled by hypothalamic stem cells, partially through the release of exosomal miRNAs.
