Ultimately, it is game of Prana! Wound-healing is mother nature’s love for you! And it works best when you are with her!
Try it. If you are aging prematurely, take a break of 15 days and spend time in jungle, near flowing river and I bet, you will start feeling younger, skin will glow and wrinkles will go away!
Modern medical science has yet to understand the word प्राण & प्राण विद्या
Mother Nature + Social community living = 100 years of healthy living for most (barring exceptions of karmic influence)
Living in a stimulating environment has a wide range of health benefits in humans and has even been shown to fight cancer in mice, but the underlying mechanisms have been unclear. A study now reveals that cognitive stimulation, social interactions, and physical activity increase lifespan in mice with colon cancer by triggering the body’s wound repair response.
“The bottom line is that there are many benefits with minimal risks to reducing stress through mind-body interventions,” says senior author Melinda Angus-Hill of the Huntsman Cancer Institute at the University of Utah. “However, more research is essential to define whether mind-body interventions drive a wound repair response in colon tumorigenesis in humans.”
Research
https://www.cell.com/cell-reports/fulltext/S2211-1247(17)30481-3?_returnURL=http%3A%2F%2Flinkinghub.elsevier.com%2Fretrieve%2Fpii%2FS2211124717304813%3Fshowall%3Dtrue
Environmental Enrichment Induces Pericyte and IgA-Dependent Wound Repair and Lifespan Extension in a Colon Tumor Model
Highlights
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Environmental enrichment (EE) enhances the lifespan of colon-tumor-bearing mice
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EE activates nuclear hormone receptor signaling commonly involved in wound repair
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EE resolves the wound repair process in tumors through IgA and pericyte activities
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EE normalizes gut microbiota and improves microbe biodiversity
Summary
Environmental enrichment (EE) replicates mind-body therapy by providing complex housing to laboratory animals to improve their activity levels, behavior, and social interactions. Using a Tcf4Het/+ ApcMin/+-mediated model of colon tumorigenesis, we found that EE vastly improved the survival of tumor-bearing animals, with differential effect on tumor load in male compared to female animals. Analysis of Tcf4Het/+ ApcMin/+ males showed drastically reduced expression of circulating inflammatory cytokines and induced nuclear hormone receptor (NHR) signaling, both of which are common in the wound repair process. Interestingly, EE provoked tumor wound repair resolution through revascularization, plasma cell recruitment and IgA secretion, replacement of glandular tumor structures with pericytes in a process reminiscent of scarring, and normalization of microbiota. These EE-dependent changes likely underlie the profound improvement in survival of colon-tumor-bearing Tcf4Het/+ ApcMin/+ males. Our studies highlight the exciting promise of EE in the design of future therapeutic strategies for colon cancer patients.
