You might not cook with it, but you almost certainly eat or use palm oil.
Palm oil is the most widely consumed vegetable oil on the planet, and it is in about half of all packaged products sold in the supermarket. While palm oil is the most efficient source of vegetable oil, its rapid expansion threatens some of the planet’s most important and sensitive habitats.
Many products that use palm oil aren’t clearly labeled. Palm oil and its derivatives can appear under many names, including:
INGREDIENTS: Vegetable Oil, Vegetable Fat, Palm Kernel, Palm Kernel Oil, Palm Fruit Oil, Palmate, Palmitate, Palmolein, Glyceryl, Stearate, Stearic Acid, Elaeis Guineensis, Palmitic Acid, Palm Stearine, Palmitoyl Oxostearamide, Palmitoyl Tetrapeptide-3, Sodium Laureth Sulfate, Sodium Lauryl Sulfate, Sodium Kernelate, Sodium Palm Kernelate, Sodium Lauryl Lactylate/Sulphate, Hyrated Palm Glycerides, Etyl Palmitate, Octyl Palmitate, Palmityl Alcohol
CONTAINS: Palm oil
Do we realize the havoc we invite @ Indonesia by increasing demand of palm oil?
The establishment of vast monoculture oil plam plantations has a number of environmental impacts.
The two most serious are:
large-scale forest conversion
loss of critical habitat for endangered species
Other impacts include:
soil erosion
air pollution
soil & water pollution
climate change
What can I do personally or at family level?
Reduce palm oil usage. Google and find out different use of palm oil.
This was about Environmental Cancer!
Recent study finds that it also helps spreading cancer in body!
Research
GROUND-BREAKING EVIDENCE THAT CANCER SPREAD IS INCREASED BY A HIGH FAT DIET AS RESEARCHERS DISCOVER NEW CANCER-SPREADING PROTEIN
A study partly funded by UK charity Worldwide Cancer Research and headed by Professor Salvador Aznar Benitah, at the Institute for Research in Barcelona (IRB) have identified for the first time a specific protein called CD36 on cancer cells which have the ability to metastasize (spread). CD36, found in the cell membranes of tumour cells, is responsible for taking up fatty acids. This unique CD36 activity and dependence on fatty acids distinguishes metastasis-initiating cells from other tumour cells. The work was published today in the leading scientific journal Nature.
They went on to test a specific saturated fatty acid called palmitic acid – a major component of animal and vegetable fats and present at high levels in palm oil which is used in many house hold products from peanut butter and processed food to toothpaste. The researchers treated human oral tumours with palmitic acid for two days then injected them into mice fed a standard diet. The team observed that all the mice with CD36 developed cancer spread compared to only half when not treated with palmitic acid.
“In mice inoculated with human tumour cells, there appears to be a direct link between fat intake and an increase in metastatic potential through CD36. More studies are needed to unravel this intriguing relationship, above all because industrialised countries are registering an alarming increase in the consumption of saturated fats and sugar,” warns Professor Benitah. “Fat is necessary for the function of the body, but uncontrolled intake can have an effect on health, as already shown for some tumours such as colon cancer, and in metastasis, as we demonstrate here.”
https://www.worldwidecancerresearch.org/news/ground-breaking-evidence-cancer-spread-increased-high-fat-diet-researchers-discover-new-cancer-spreading-protein/
Targeting metastasis-initiating cells through the fatty acid receptor CD36
The fact that the identity of the cells that initiate metastasis in most human cancers is unknown hampers the development of antimetastatic therapies. Here we describe a subpopulation of CD44bright cells in human oral carcinomas that do not overexpress mesenchymal genes, are slow-cycling, express high levels of the fatty acid receptor CD36 and lipid metabolism genes, and are unique in their ability to initiate metastasis. Palmitic acid or a high-fat diet specifically boosts the metastatic potential of CD36+ metastasis-initiating cells in a CD36-dependent manner. The use of neutralizing antibodies to block CD36 causes almost complete inhibition of metastasis in immunodeficient or immunocompetent orthotopic mouse models of human oral cancer, with no side effects. Clinically, the presence of CD36+ metastasis-initiating cells correlates with a poor prognosis for numerous types of carcinomas, and inhibition of CD36 also impairs metastasis, at least in human melanoma- and breast cancer-derived tumours. Together, our results indicate that metastasis-initiating cells particularly rely on dietary lipids to promote metastasis.
http://www.nature.com/nature/journal/vaop/ncurrent/full/nature20791.html
