I have been saying this for long based on experience and observations. Microbes are nothing but manifestation of our bodily Prana. They are not organized as organ but do work as community with special purpose to serve the body. And since they are manifested based on Prana, they can be controlled by Pranic exercises. Asana(s) and specifically Surya Namaskar plays vital role here for their well-designed postures and bends with regulated breathing.
Exercise changes gut microbial composition independent of diet, team reports
All the bends that one performs during Surya Namaskar, work as churning process for Prana. And it directly affects the composition of microbes in GUT, GUT being Muladhara chakra.
Probiotic food is not the primary medicine. Exercise is. Surya Namaskar – Must must!
Even playing hockey helps a lot for its natural bending posture that gives enough churning in muladhra.
Research
Exercise changes gut microbial composition independent of diet, team reports
https://news.illinois.edu/blog/view/6367/586206
Two studies – one in mice and the other in human subjects – offer the first definitive evidence that exercise alone can change the composition of microbes in the gut. The studies were designed to isolate exercise-induced changes from other factors – such as diet or antibiotic use – that might alter the intestinal microbiota.
In the first study, scientists transplanted fecal material from exercised and sedentary mice into the colons of sedentary germ-free mice, which had been raised in a sterile facility and had no microbiota of their own. In the second study, the team tracked changes in the composition of gut microbiota in human participants as they transitioned from a sedentary lifestyle to a more active one – and back again.
“These are the first studies to show that exercise can have an effect on your gut independent of diet or other factors,” said Jeffrey Woods, a University of Illinois professor of kinesiology and community health who led the research with former doctoral student Jacob Allen, now a postdoctoral researcher at Nationwide Children’s Hospital in Columbus, Ohio. The work with mice was conducted at the U. of I. and with scientists at the Mayo Clinic in Rochester, Minnesota, who develop and maintain the germ-free mice. The work in humans was conducted at Illinois.
Exercise training-induced modification of the gut microbiota persists after microbiota colonization and attenuates the response to chemically-induced colitis in gnotobiotic mice
http://www.tandfonline.com/doi/full/10.1080/19490976.2017.1372077
Exercise reduces the risk of inflammatory disease by modulating a variety of tissue and cell types, including those within the gastrointestinal tract. Recent data indicates that exercise can also alter the gut microbiota, but little is known as to whether these changes affect host function. Here, we use a germ-free (GF) animal model to test whether exercise-induced modifications in the gut microbiota can directly affect host responses to microbiota colonization and chemically-induced colitis. Donor mice (n = 19) received access to a running wheel (n = 10) or remained without access (n = 9) for a period of six weeks. After euthanasia, cecal contents were pooled by activity treatment and transplanted into two separate cohorts of GF mice. Two experiments were then conducted. First, mice were euthanized five weeks after the microbiota transplant and tissues were collected for analysis. A second cohort of GF mice were colonized by donor microbiotas for four weeks before dextran-sodium-sulfate was administered to induce acute colitis, after which mice were euthanized for tissue analysis. We observed that microbial transplants from donor (exercised or control) mice led to differences in microbiota β-diversity, metabolite profiles, colon inflammation, and body mass in recipient mice five weeks after colonization. We also demonstrate that colonization of mice with a gut microbiota from exercise-trained mice led to an attenuated response to chemical colitis, evidenced by reduced colon shortening, attenuated mucus depletion and augmented expression of cytokines involved in tissue regeneration. Exercise-induced modifications in the gut microbiota can mediate host-microbial interactions with potentially beneficial outcomes for the host.
Exercise Alters Gut Microbiota Composition and Function in Lean and Obese Humans
https://insights.ovid.com/crossref?an=00005768-900000000-97045
Abstract
PURPOSE
Exercise is associated with altered gut microbial composition, but studies have not investigated whether the gut microbiota and associated metabolites are modulated by exercise training in humans. We explored the impact of six weeks of endurance exercise on the composition, functional capacity, and metabolic output of the gut microbiota in lean and obese adults with multiple-day dietary controls prior to outcome variable collection.
METHODS
Thirty-two lean (n=18 [9 female]) and obese (n=14 [11 female]), previously sedentary subjects participated in six weeks of supervised, endurance-based exercise training (3 days/wk) that progressed from 30 to 60 minutes/day and from moderate (60% of heart rate reserve [HRR]) to vigorous intensity (75% HRR). Subsequently, participants subsequently returned to a sedentary lifestyle activity for a six week washout period. Fecal samples were collected before and after six weeks of exercise, as well as after the sedentary washout period, with 3-day dietary controls in place prior to each collection.
RESULTS
β-diversity analysis revealed that exercise-induced alterations of the gut microbiota were dependent on obesity status. Exercise increased fecal concentrations of short chain fatty acids (SCFAs) in lean, but not obese, participants. Exercise-induced shifts in metabolic output of the microbiota paralleled changes in bacterial genes and taxa capable of SCFA production. Lastly, exercise-induced changes in the microbiota were largely reversed once exercise training ceased.
CONCLUSION
These findings suggest that exercise training induces compositional and functional changes in the human gut microbiota that are dependent on obesity status, independent of diet and contingent on the sustainment of exercise.
