We are groomed to consider Viruses and Bacteria as enemy. So idiotic form of science
Virus is a piece of nucleic acid (DNA or RNA) wrapped in a thin coat of protein. DNA or RNA is a packet of information exchanged among cells or live organism, just like data is exchanged between two computers using network and based on data exchange, computers act. Similarly, based on information exchange, cells act.
Packet of information is useless if there are no software programs written to interpret the data and commands. If software has bugs, wrong interpretation can happen and computer can give wrong output.
Similarly, virus is harmless if cells are programmed to interpret message correctly.
So where is the issue with Ebola or any other virus? They are harmless. Issue is with cells. Under stress, cells sometimes do not act as per programs. Or in other words, they raises false alarms by producing more specific DNA packets i.e. Viruses.
Culprit is not dead DNA strips generated by cells but cell level stress.
Solution is not to subside viral fever or take flu vaccines. Solution is to reduce cell level stress.
Read this: your body cells are actually helping viruses to let in and execute the code.
Viruses are dead messages. They have critical information about intra-cellular, intra-body environmental condition. And so cells do not see them enemy.
Correct viral infection medicine is not to purge dead messages but to reduce cellular, environmental stress i.e. Increase प्राण. Decrease physical and mental stress. Take immune booster natural medicines (lemon, turmeric etc) and you are good.
Co-option of Membrane Wounding Enables Virus Penetration into Cells
During cell entry, non-enveloped viruses undergo partial uncoating to expose membrane lytic proteins for gaining access to the cytoplasm. We report that adenovirus uses membrane piercing to induce and hijack cellular wound removal processes that facilitate further membrane disruption and infection. Incoming adenovirus stimulates calcium influx and lysosomal exocytosis, a membrane repair mechanism resulting in release of acid sphingomyelinase (ASMase) and degradation of sphingomyelin to ceramide lipids in the plasma membrane. Lysosomal exocytosis is triggered by small plasma membrane lesions induced by the viral membrane lytic protein-VI, which is exposed upon mechanical cues from virus receptors, followed by virus endocytosis into leaky endosomes. Chemical inhibition or RNA interference of ASMase slows virus endocytosis, inhibits virus escape to the cytosol, and reduces infection. Ceramide enhances binding of protein-VI to lipid membranes and protein-VI-induced membrane rupture. Thus, adenovirus uses a positive feedback loop between virus uncoating and lipid signaling for efficient membrane penetration.