Note: This post is not written to discourage readers doing regular exercise 🙂 but to suggest variation in exercise based on your innate prakriti and season of the year.
You believe in your जाति or not, but it will remain with you and your kids for at least 7 generations. Where you live, what you eat, what trauma you face, and same events for your grandparents and great-parents up to 7 generations, matter a lot.
जाति/वर्ण is your identity. It can serve you best in intelligent secular society. Based on your जाति/वर्ण, you receive food, shelter and rituals.
There is no discrimination here.
Now read this modern research in this context.
“Arrhythmogenic ventricular cardiomyopathy (AVC) is the most common heart condition that causes sudden cardiac death during intense exercise. ”
If your body constitution is like shudra or khstriya, you certainly will sustain Endurance exercise best. But if you ae Vaishya or Brahman by वर्ण, your body is not designed for endurance exercise as it is for Shudra and Kshtriya! You are inviting unforeseen consequences!
Get real. Respect your body. Act as per your innate nature. And to learn to act is the motive of life! 🙂 (Ideally by end of Brahmcharya AShram, it becomes clear)
Like same food everyone, same exercise for everyone, is a silent killer.
When Exercise Is Unhealthy For The Heart: Researchers Describe How Heart Problems And Sudden Cardiac Death Occur With Endurance Exercise
Endurance exercise accelerates the development of heart problems in individuals with a particular genetic mutation, a new study in American Journal of Physiology—Heart and Circulatory Physiologyreports.
Arrhythmogenic ventricular cardiomyopathy (AVC) is the most common heart condition that causes sudden cardiac death during intense exercise. The study found that in mice with a mutated version of desmoplakin, a protein that helps maintain the structure of the heart wall, exercise made the heart walls come apart sooner, leading to earlier development of AVC. According to lead researcher Jeffrey Towbin, MD, the findings confirm the role of the mutation in the development of AVC and offer insight into how to best manage exercise in individuals with AVC, which currently has no standardized strategy.
The cells in the heart wall are stacked in interlocking layers like bricks in a brick wall. They are attached to each other by links called desmosomes that dot the surface of the cells. Exercise can overstretch the heart wall, but these links keep the cells from disconnecting from each other and the heart wall from coming apart. Desmosomes are made up of several proteins, including desmoplakin. Defects in the proteins, as in AVC, weaken the cell-cell links. The cells do not withstand the extra stretch from exercise and detach from each other. Scar tissue fills the gaps between the disconnected cells, further reducing the heart wall’s ability to withstand increases in workload such as exercise.
Several studies observed that mice with a mutated form of desmoplakin developed the same heart problems seen in patients with AVC. In this new study, researchers at the Cincinnati Children’s Hospital Medical Center discovered endurance exercise led to earlier onset of AVC symptoms in mice with the mutated desmoplakin. Moreover, the desmosomes started coming apart before changes in heart function were detected. Exercise also changed the signaling in the molecular pathway that promoted growth of new cells and prevented deposition of fat, the Wnt-β-catenin pathway, offering an explanation for the accelerated development of AVC.